2. 您的位置
3. 首页
4. 会议论文
5. INHIBITION OF KAP1 ENHANCES HYPOXIA-INDUCED KAPOSIS SARCOMAASSOCIATED HERPESVIRUS REACTIVATION THROU

INHIBITION OF KAP1 ENHANCES HYPOXIA-INDUCED KAPOSIS SARCOMAASSOCIATED HERPESVIRUS REACTIVATION THROU

来源 :17th International Workshop on Kaposi’s Sarcoma Associated H | 被引量 : 0次 | 上传用户：zhangxueyh

【摘 要】

：

Hypoxia-inducible factor 1alpha (HIF-1alpha) has been frequently implicated in many cancers as well as viral pathogenesis.Kaposis sarcoma-associated herpesvirus

【作 者】

：

Liming Zhang Caixia Zhu Yi Guo Fang Wei Jie Lu Jing Qin Shuvomoy Banerjee Junwen Wang Hong Shang Subhash C.Verma Zhenghong Yuan Erle S.Robertson Qiliang Cai 

【机 构】

：

MOE&MOHKeyLaboratoryofMedicalMolecularVirology,SchoolofBasicMedicine,ShanghaiMedicalCollege,FudanUni

【出 处】

：

17th International Workshop on Kaposi’s Sarcoma Associated H

【发表日期】

：

2014年7期

下载到本地 , 更方便阅读

下载此文 赞助VIP

声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架

论文部分内容阅读

Hypoxia-inducible factor 1alpha (HIF-1alpha) has been frequently implicated in many cancers as well as viral pathogenesis.Kaposis sarcoma-associated herpesvirus (KSHV) is linked to several human malignancies.It can stabilize HIF-1alpha during latent infection and undergoes lytic replication in response to hypoxic stress.

其他文献

No contribution of IP3-R(2) to disease phenotype in models of dilated cardiomyopathy or pressure ove

Background: We investigated the contribution of inositol(1,4,5)trisphosphate (Ins(1,4,5)P3,IP3) receptors (IP3-R) to disease progression in mouse models of dila

会议

Marcching to the Beat of the Calcium Drum Arrhythmia Mechanisms in Gain-and Loss-of-Function Ryanodi

Type 2 ryanodine receptors (RyR2s) are the calcium release channels of sarcoplasmic reticulum (SR) that provide the majority of calcium ions (Ca2+) necessary to

会议

Lack and presence of Ca2+-activated Cl-channels dominates the differential effects of Ca2+ sparks on

Ca2+ sparks induce relaxation of cerebral artery smooth muscle cells (CASMCs) and contraction of airway smooth muscle cells (ASMCs);however, a comparative inves

会议

cerebral artery smooth muscle cellsairway smooth muscle cellsCa2+sparksSTICs

Dynamic Ca2+ sensitivity of BK channels

Large-conductance calcium-activated K+ (BK-type, mSlo1 or mutations) channels, abundantly distributed in the excitable cells, response to the intracellular Ca2+

会议

RHADINOVIRAL TEGUMENT PROTEINS ARE CRITICAL FOR THE ANTAGONIZATION OF ND10-1NSTITUTED INTRINSIC IMMU

Nuclear domain 10 (ND10) components are restriction factors that inhibit herpesviral replication.Effector proteins of different herpesviruses can antagonize thi

会议

THE INHIBITION OF MURINE GAMMAHERPESVIRUS-68 LYTIC REPLICATION IN MACROPHAGES BY INTERFERON GAMMA IS

Interferon gamma (IFN-Y) as a type Ⅱ interferon was revealed to have multiple effects on murine gamma herpesvirus-68, a rodent gamma herpesvirus closely relate

会议

EPHA2 IS A HOST FACTOR FOR EPSTEIN-BARR VIRUS INFECTION OF NASOPHARYNGEAL EPITHELIAL CELLS

Epstein-Barr virus (EBV) has been causally associated with lymphoid and epithelial malignancies, suggesting its primary tropisms for these two types of cells.Th

会议

FLUORESCENT TAGGING AND CELLULAR DISTRIBUTION OF THE KAPOSIS SARCOMAASSOCIATED HERPESVIRUS (KSHV) OP

Kaposis sarcoma-associated herpesvirus (KSHV), also known as human herpesvirus 8 (HHV-8), is a cancer-related human virus, classified as a member of the gammahe

会议

THE STIMULATOR OF INTERFERON GENES PLAYS AN IMPORTANT ROLE IN KSHV LIFE CYCLE

The stimulator of interferon genes (STING;also known as TMEM173, MPYS, MITA and ERIS) is a critical signaling molecule in innate immune response to cytosolic nu

会议

HERPESVIRAL G PROTEIN-COUPLED RECEPTORS SHORTCUT NFAT ACTIVATION TO INDUCE TUMOR FORMATION VIA TARGE

G protein-coupled receptors (GPCRs) constitute the largest family of proteins that transmit signal to regulate an array of fundamental biological processes.Viru

会议