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目的:探讨小凹蛋白-1(Cav-1)上调人脐静脉内皮细胞(HUVECs)钙敏感受体(CaR)介导一氧化氮(NO)生成的作用机制。方法:培养HUVECs,取同代细胞随机分为:对照组;CaR激动剂(精胺)组;小凹(caveolae)结构破坏剂(filipin,1.5
AIM: To investigate the mechanism of caveolin-1 (Cav-1) upregulation of nitric oxide (NO) mediated by calcium sensitive receptor (CaR) in human umbilical vein endothelial cells (HUVECs). Methods: HUVECs were cultured and their offspring were randomly divided into control group, CaR agonist (spermine) group, caveolae structural disruptor