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Objectives To investigate the effects of EGB on advanced glycosylation end products (AGEs)-induced cardiomyocyte injury, and the role of endoplasmic reticulum stress (ERS) in the process.Methods Cultured neonatal rat cardiomyocytes were randomly divided into four groups: control, EGB, AGEs, and AGEs + EGB.MTT assay was used to measure cell viability.