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Background: There is increasing evidence that cytoskeleton remodeling is involved in cancer progression.Wiskott-Aldrich syndrome Protein(WASP)family represents a key regulator of actin cytoskeleton remodeling.However,the underlying mechanism of WASP family in cancer development has remained elusive.Here,we studied the role of WASP and SCAR Homolog(WASH),a recently identified WASP family member,in human esophageal squamous cell carcinoma(ESCC).