Deficiency of α-tubulin acetylation increases microtubule dynamics

来源 :The 6th FAONS Congress and 11th Biennial Conference of CNS(第 | 被引量 : 0次 | 上传用户:gb1107
下载到本地 , 更方便阅读
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
  Objective Microtubules are hollow cylinder filaments assembled from α/β-tubulin heterodimers in eukaryotes.
其他文献
Autism is a developmental disorder with characteristic deficiency in social behavior and communication skills and the presence of stereotypical behavior.
Parkinsons disease(PD)is a chronic progressive neurodegenerative movement disorder characterized by a profound and selective loss of nigrostriatal dopaminergic neurons.
Various recent studies showed that postsynaptic density(PSD)scaffold proteins Shank involved in autism,schizophrenia and Alzheimers disease.
Althoughpharmacological dopamine replacement therapy and deep brain electrical stimulation(DBS)can alleviate the motor symptoms of Parkinson disease(PD),side effects of drugs and a small fraction of p
The precision of axon pathfinding is critical for the neocortex development and the formation of the neural circuitry.
Objective Axonal localization of mRNAs and translation are important for axon elongation.Fmr1-encoded protein,FMRP,as a RNA-binding protein(RBP),has been suggested playing a central role in dendritic
Spinster homolog 2(SPNS2)is the only membrane transporter that has been demonstrated to secrete sphingosine-1-phosphate(S1P)in vivo.
To date,there are no published reports of the profile of microRNA(miRNA),long non-coding RNA(lncRNA)and messenger RNA(mRNA)related to delayed motherhood or a comparison of the delayed motherhood-speci
NG2-expressing cells in CNS consist of heterogeneous cell populations.
Its accepted that amyloid β-derived diffusible ligands(ADDLs)play a prominent role in triggering the early cognitive deficits that constitute Alzheimers disease(AD).