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5. Cathepsin S activity controls injury-related vascular repair in mice via the p38MAPK and PI3K-Akt/p-

Cathepsin S activity controls injury-related vascular repair in mice via the p38MAPK and PI3K-Akt/p-

来源 :Ready for 2018- the 4th China International Forum on Hyperte | 被引量 : 0次 | 上传用户：lingyuehqu2009

【摘 要】

：

　　Background:Neointimal hyperplasia is a major cause of restenosis after coronary intervention.The role of cathepsin S (CatS) in endovascular treatment injury

【作 者】

：

HongxianWu[1]XianwuCheng[2]KyosukeTakeshita[3]LinaHu[4]ChenHu[5]QiunaDu[6]ZheHuang[7]XiaohongZhang[5]MasafumiKuzuya[4]GuopingShi[8]QiuyanDai[6]ToyoakiMurohara[3] 

【机 构】

：

Department of Cardiology,Nagoya University Graduate School of Medicine,Nagoya,Japan

【出 处】

：

Ready for 2018- the 4th China International Forum on Hyperte

【发表日期】

：

2015年期

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　　Background:Neointimal hyperplasia is a major cause of restenosis after coronary intervention.The role of cathepsin S (CatS) in endovascular treatment injury-induced neointimal hyperplasia is poorly understood.Our findings described herein revealed that CatS has a role in injury-related vascular repair in mice.

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