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Introduction: Despiteadvancesinsurgicalrepairtechniques,healingofflexortendonsremainsproblematicduetopoorvascularizationandhypocellularity[1,2].Whilelocaltenocytesplayadominantroleinintrinsictendonhealing,themechanismthatinitiatescellproliferationandmigrationaftertendoninjuryisnotwellunderstood.