论文部分内容阅读
Objectives Chronic inflammation have emerged as important role in heart failure (HF).However, suppressing local inflammatory effect of ginsenoside Rb1 (Gs-Rb1) in HF remains to be elucidated.Thus, the present study aimed at examining the effect of Gs-Rb1 in proinflammatory factor, such as tumour necrosis factor alpha (INFα), nuclear transcription factor kappa B (NF-κB) and interleukin (IL) basing on rat HF model and cardiomyocyte insult induced by adriamycin, in vivo and ex vivo.