ICR小鼠受致死量射线照射以后,接受有机锗治疗14天.剂量为500mg/kg的小鼠30天活存率比对照组提高37.5％;当Ce—132剂量为500mg/kg和1000mg/kg时,其内源性脾结节的数量明显地高于对照组。实验结果表明,Ce—132具有一定的治疗急性骨髓型放射病的作用和刺激内源性造血干细胞的增殖与分化作用。 After ICL mice were exposed to lethal doses of radiation, they were treated with organic germanium for 14 days. The 30-day survival rate of the mice at a dose of 500 mg/kg was 37.5% higher than that of the control group; when the Ce-132 dose was 500 mg/kg and 1000 mg/kg At the time, the number of endogenous splenic nodules was significantly higher than that of the control group. The experimental results show that Ce-132 has a certain role in the treatment of acute bone marrow-type radiation sickness and stimulate the proliferation and differentiation of endogenous hematopoietic stem cells.