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蛛网膜下腔出血(SAH)是常见的急性脑血管病之一。脑血管痉挛(CVS)是蛛网膜下腔出血后常见的严重并发症,其严重影响患者的预后,大大增加了此病的病死率和致残率。因而,一直是蛛网膜下腔出血基础研究和临床治疗的重点和难点。迄今为止,对SAH后CVS发生、发展的病因和机制尚不十分清楚,大多数学者认为是一个多因素复杂的过程,其中NO在SAH后CVS中的作用日益受到重视。现将NO及其在SAH后CVS
Subarachnoid hemorrhage (SAH) is one of the common acute cerebrovascular diseases. Cerebral vasospasm (CVS) is a common serious complication after subarachnoid hemorrhage, which seriously affects the prognosis of patients and greatly increases the mortality and morbidity of the disease. Therefore, it has been the focus and difficulty of basic research and clinical treatment of subarachnoid hemorrhage. So far, the etiology and mechanism of CVS after SAH is not yet clear, most scholars think is a multi-factor complex process, in which the role of NO in CVS after SAH is increasingly valued. Now NO and its CVS after SAH