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持续3h电刺激,蟾蜍腓肠肌肌肉力量降至起始肌力的10%以下,并可诱发延迟性肌肉结构异常。刺激后3h,超微结构可见大量小收缩带、异常Z 带及局灶性肌丝紊乱。至刺激后24h,变化可发展为大面积肌原纤维变性。经斜刺处理,肌力即刻上升,针后3h,收缩带消失,Z 带变化显著减轻:至针后24h,肌肉结构已基本恢复正常。电刺激至力竭的离体半腱肌,在任氏液中针刺后,肌力恢复加快。如去掉任氏液中Ca~(2+),部分抑制细胞内Ca外排后,针刺效应则显著下降。结果提示,针刺很可能通过某种细胞膜转运机制降低细胞内Ca~(2(?)),迅速缓解肌痉挛和Z 带异常,并促进变化肌丝和肌力加快恢复。
After 3 hours of electrical stimulation, the muscle strength of toad gastrocnemius muscle was reduced to less than 10% of the initial muscle strength, and delayed muscle structural abnormalities could be induced. 3h after stimulation, ultrastructure showed a large number of small contraction zone, abnormal Z zone and focal myofilament disorders. 24h after stimulation, the change can develop into large-area myofibrillar degeneration. The diastolic process, muscle strength immediately, 3h after the needle, contraction zone disappeared, Z zone significantly reduced: up to 24h after the needle, the muscle structure has returned to normal. Electrical stimulation to exhaustion of the isolated semitendinosus muscle, Ren Ren liquid acupuncture, muscle strength recovery accelerated. Such as Ren Ren solution removed Ca ~ (2+), partial inhibition of intracellular Ca efflux, acupuncture effect was significantly decreased. The results suggest that acupuncture is likely to reduce intracellular Ca ~ (2 (?)) Via some cell membrane transport mechanism, quickly relieve muscle spasms and abnormal Z band, and promote the recovery of myofilament and muscle strength.