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肝缺血再灌注时组织细胞发生一系列代谢、结构和功能的损伤 ,甚至导致肝功能衰竭。氧自由基损伤可能是缺血再灌注肝损伤的重要机制之一。本实验采用大鼠肝缺血再灌注模型 ,观察血清脂质过氧化物(MDA)与超氧化物歧化酶 (SOD)的变化及其临床意义 ,为临床防治肝缺血再灌注损伤提供
Tissue cells undergo a series of metabolic, structural and functional injuries during hepatic ischemia-reperfusion and even lead to liver failure. Oxygen free radical injury may be one of the important mechanisms of ischemia-reperfusion liver injury. In this study, rat liver ischemia-reperfusion model was used to observe the changes of serum lipid peroxidation (MDA) and superoxide dismutase (SOD) and its clinical significance for the prevention and treatment of liver ischemia-reperfusion injury