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心肌缺血并不意味着心肌已经发生了不可逆性坏死 ,近来证实缺血心肌组织中至少存在三种重要的缺血性适应 :心肌冬眠、心肌顿抑和缺血预适应。心肌冬眠是心肌对长期低血流灌注做出的心脏收缩减弱的反应状态。心肌顿抑是短暂缺血血流恢复后仍存留的心肌收缩功能障碍的现象。缺血预适应是一种内源性的自我保护机制 ,是指反复缺血后心肌对缺血的耐受力增加的现象。冬眠和顿抑心肌仍然是存活的 ,应及早识别并采取积极的措施 ,挽救这部分尚存活的心肌 ,尽可能发挥缺血预适应的心肌保护作用 ,改善缺血性心脏病患者的预后
Myocardial ischemia does not mean that the myocardium has undergone irreversible necrosis. It has recently been demonstrated that there are at least three important ischemic adaptations in ischemic myocardium: myocardial hibernation, myocardial stunning, and ischemic preconditioning. Myocardial hibernation is a state of response to the diminished cardiac contractions of the myocardium caused by long-term hypoperfusion. Myocardial dyssynchrony is a phenomenon of myocardial systolic dysfunction remaining after transient ischemia and blood flow recovery. Ischemic preconditioning is an endogenous self-protection mechanism, refers to the repeated ischemia of myocardial ischemia increased tolerance phenomenon. Hibernation and stunned myocardium are still alive, and should be identified and taken as soon as possible to take active measures to save this part of the surviving myocardium, as far as possible to play the role of myocardial ischemic preconditioning and improve the prognosis of patients with ischemic heart disease