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越来越多的研究表明,微丝骨架参与植物先天免疫过程,但是其作用机制尚不明确.本研究发现,棉花(Gossypium spp.)的profilin基因(GhPFN2)在大丽轮枝菌(Verticillium dahliae)侵染条件下表达水平上调,推测GhPFN2可能参与棉花应答黄萎病过程.当棉花根部侵染大丽轮枝菌后,根表皮细胞中微丝的密度和成束度显著增加.与野生型相比,过量表达GhPFN2棉花对黄萎病的耐受性提高,并且棉花根部微丝骨架高级结构与野生型受到大丽轮枝菌侵染后的表型一致.这些结果表明,GhPFN2能够介导微丝骨架的重排,进而参与棉花抵御大丽轮枝菌的侵染过程.
More and more researches have shown that actin filaments are involved in plant innate immunity, but its mechanism of action is not yet clear.In this study, we found that the profilin gene (GhPFN2) of cotton (Gossypium spp.) Was expressed in Verticillium dahliae ), Suggesting that GhPFN2 may be involved in the process of cotton response to Verticillium wilt.When cotton roots were infected with Verticillium dahliae, the density and bundles of microfilaments in root epidermal cells increased significantly.Compared with wild type , The tolerance of the over-expressed GhPFN2 cotton to Verticillium wilt was increased and the higher structure of the microfilament backbone in cotton roots was consistent with the phenotype of the wild type infected by V. daurica.These results indicated that GhPFN2 could mediate Microfilament rearrangement, and then involved in cotton to resist the infection process of Verticillium dahlia.