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目的观察45天-6°头低位卧床模拟失重条件对个体返回抑制能力产生的影响。方法分别在卧床前第6～4天,卧床第11～13天、第20～22天、第32～34天、第40～42天和卧床后第8～10天采用线索提示范式测试16名男性个体的返回抑制能力。结果刺激始发异步性(SOA)与线索有效性交互作用不显著,说明个体在现有SOA条件下没有出现返回抑制现象。SOA主效应接近边缘显著,个体在SOA为900 ms的反应时显著小于为1200 ms的反应时,且在卧床第11～13天,第20～22天以及卧床后恢复阶段个体在不同SOA条件下的反应时差别消失,说明卧床前期和卧床后恢复阶段个体抑制能力受到了影响,而卧床中期抑制能力逐渐恢复。结论 45天-6°头低位卧床可能延长了个体的认知加工时间,导致产生返回抑制现象需要的SOA延长;卧床条件对个体抑制能力产生了影响,个体在卧床前期以及卧床后恢复阶段的抑制能力受到影响,表现出由受损到适应恢复到受损的变化趋势。
Objective To observe the effects of 45 days -6 ° head-on-bed simulated weightlessness on the return inhibition of individuals. Methods in the sixth to 4 days before the bed, the bed of 11 to 13 days 20 to 22 days 32 to 34 days 40 to 42 days after the first bed 8 to 10 days using 16 test paradigm Cues Male individual’s ability to suppress depression. Results There was no significant interaction between stimulatory asynchrony (SOA) and clue validity, indicating that individuals did not exhibit repression under the existing SOA conditions. SOA main effect close marginally significant, individuals SOA when 900 ms reaction is significantly less than for the 1200 ms the reaction, and the bed day 11 to 13, 20 to 22 days, and after bed recovery phase individuals under SOA Conditions Of the reaction time difference disappear, indicating that bed rest and bed rest phase inhibition of individuals affected, and bed restitution capacity gradually restored. Conclusion -6 ° head-down 45 days may be extended bed rest individual cognitive processing time, resulting in suppression needed to return to extend the SOA; ability to inhibit individual bed conditions had an impact, and a bed of inhibiting early in the recovery phase after bed Ability to be affected, showing a change from damage to recovery to damage.