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为从组织形态学角度评价幽门螺杆菌(HP)对胃酸分泌的影响,以231例胃体、胃窦及十二指肠粘膜活检病例为研究对象,探讨十二指肠胃化生(GM)与HP相关性慢性胃炎的感染部位、炎症活动度及萎缩之间的关系.结果表明HP在胃窦感染为主时十二指肠GM的发生增加;在冒体感染为主时则相反.十二指肠GM的程度与胃窦炎的活动度成正相关,与胃体炎活动度成负相关;胃窦粘膜萎缩时HP感染可减少十二指肠GM的发生.本文结果提示:HP在胃窦感染时能促进胃酸的分泌,而在胃体感染或胃窦粘膜萎缩时则可抑制胃酸分泌.
In order to assess the effect of Helicobacter pylori (HP) on gastric acid secretion from the perspective of histomorphology, 231 cases of gastric, gastric and duodenal mucosal biopsy were studied to investigate the relationship between duodenal gasification (GM) HP-related chronic gastritis infection site, the relationship between the degree of inflammatory activity and atrophy.The results show that HP mainly in the antrum of the duodenum increased when the occurrence of GM; when the host-based infection is the opposite. The degree of intestine GM was positively correlated with the activity of gastric antrum and negatively correlated with the activity of gastritis.HP infection in the antral antral mucosa could reduce the occurrence of duodenal GM.The results of this paper suggest that HP is more effective in the antrum Infection can promote the secretion of gastric acid, gastric mucosal infection or gastric atrophy can inhibit gastric acid secretion.