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脑梗塞慢性期同侧非梗塞的大脑脚发生萎缩,推测是由于锥体系的Waller 变性引起。近年来还发现大脑中动脉区的脑梗塞,虽丘脑没有直接缺血但在缺血的同侧出现慢性进行的丘脑萎缩。本文报告1例右偏瘫14年的患者,MRI 发现除左放射冠,壳核的梗塞灶外,同侧丘脑、大脑脚、中脑、桥脑、小脑也有萎缩:患者71岁,男性,即往有高血压史。1976年12月12日突起右上下肢不全瘫痪,当地诊断为脑卒中,未行CT 及脑血管造影检查。经康复治疗瘫痪逐渐好转,一直服用抗凝和降压药。为排除脑内出血灶和确定进一步的药物疗法,于1990年11月15日转诊。神查:右上下肢肌力4～4~+级,轻度构音障碍,右
Cerebral infarction in the ipsilateral non-infarction of the cerebral contralateral brain atrophy occurs, presumably due to cone system Waller degeneration caused. In recent years, cerebral infarction in the middle cerebral artery region has also been found. Chronic atrophy of the thalamus appears on the ipsilateral side of the ischemia, although there is no direct ischemia in the thalamus. In this paper, we report a case of right hemiplegia for 14 years. MRI showed atrophy in the ipsilateral thalamus, cerebral peduncle, midbrain, pons and cerebellum except for the left coronavirus and the putamen. The patients were 71 years old and males, Have a history of hypertension. On December 12, 1976, the right upper extremity was incompletely paralyzed, and the local diagnosis was a stroke. CT and cerebral angiography were not performed. After rehabilitation treatment paralysis gradually improved, has been taking anticoagulants and antihypertensive drugs. Referral was November 15, 1990, to exclude intracerebral hemorrhage and to determine further pharmacotherapy. God check: right upper limb muscle strength 4 ~ 4 ~ + level, mild dysarthria, right