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本文对40例2岁至40岁的隐睾活检组织进行了生殖病理观察、定量组织学研究、免疫组织化学和隐睾细胞生物学观察,发现存在曲细精管发育不全,生精细胞发育不全,在成年隐睾尤为明显,而成年隐睾支持细胞呈增生状态。部分病例缺乏精原细胞可能是先天性的。成年隐睾中相当一部分病例的曲细精管界膜和血管壁有免疫复合物沉积,表明存在抗睾丸膜抗体存在。生精细胞、支持细胞和睾丸间质细胞对隐睾条件表现出不同的反应;生精细胞分化发育受阻,一般无精子发生;支持细胞发育受阻呈完全未成熟状态,部分受阻则形成部分成熟型支持细胞。间质细胞可发育成熟,并有亢进现象。
In this paper, 40 cases of cryptorchidism from 2 to 40 years of biopsy tissue were observed in reproductive pathology, quantitative histology, immunohistochemistry and cryptorchid cell biology found that there are seminiferous tubule hypoplasia, spermatogenic hypoplasia , Especially in adult cryptorchidism, and adult cryptorchid cells supporting cells showed hyperplasia. In some cases the lack of spermatogonia may be congenital. A considerable part of adult cryptorchidism cases of the seminiferous tubules and vascular wall immune complex deposition, indicating the presence of anti-testicular membrane antibodies. Spermatogenic cells, supporting cells and testicular stromal cells showed different responses to cryptorchidism conditions; spermatogenic cell differentiation and development were blocked, and generally no spermatogenesis; supporting cell development was completely immature state, partially blocked form part of the mature Support cells. Interstitial cells can mature, and hyperthyroidism phenomenon.