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野木瓜注射液（InjectionstauntoniaeIS）涂布于大鼠隐神经4mm长的一段上。50％IS在6min内使隐神经复合动作电位诸成分的幅度下降，潜伏期延长。尤其是AS和C成分，它们的幅度分别下降为对照的47±23．0％和44％±20％（x±SD，、n＝8，P＜0．01）。10％IS对诸成分的影响大大减弱，但Aαβ、Aδ和C成分的潜伏期依然明显延长，延长值分别为0．05±0．01ms，0．28±0．070s（n＝5，P＜0．05）和6．99±1．48ms（n＝5，P＜0．01）。IS对Aδ和C成分的影响比对Aαβ成分影响大得多，并有量效关系。IS对神经传导的阻滞作用是可逆的。
Wild Stawonia injection (Injection stauntoniaeIS) was applied to a 4 mm long section of the saphenous nerve of the rat. 50%IS decreased the amplitude of the components of the compound neural action potential of the saphenous nerve within 6 min and prolonged the incubation period. Especially for the AS and C components, their amplitudes decreased to 47±23.0% and 44%±20% of the control, respectively (x±SD, n=8, P<0.01). The effect of 10% IS on the components was greatly reduced, but the latency of Aαβ, Aδ, and C components was still significantly prolonged. The prolongation values were 0.05±0.01 ms and 0.28±0.070 s, respectively (n=5, P< 0.05) and 6.99±1.48 ms (n=5, P<0.01). The effect of IS on the Aδ and C components is much greater than that of the Aαβ component, and there is a dose-effect relationship. The retardation of nerve conduction by IS is reversible.