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充血性心力衰竭似乎能够显著地影响控制心血管功能的神经机能,表现为副交感神经作用的减退和交感神经张力的亢进.文献记载,充血性心力衰竭者不论在休息或活动情况下,血浆中去甲肾上腺素的含量都有大幅度增高,而心脏本身组织却缺乏儿茶酚胺.从人类和充血性心力衰竭的动物模型中也发现心肌细胞内的儿茶酚胺含量降低,而且交感神经所支配的血管反应性增高.与此相反,心脏迷走神经的传出冲动却减弱,由加压刺激所应有的反射性心率减慢作用明显降低.
Congestive heart failure appears to significantly affect the neural function of controlling cardiovascular function, manifested as a decrease in parasympathetic function and an increase in sympathetic tone, documented in patients with congestive heart failure either in rest or in activity Norepinephrine levels have increased significantly, while the heart itself is the lack of tissue catecholamines from human and congestive heart failure animal model also found that myocardial cell catecholamine levels decreased, and sympathetic vessels dominated vascular reactivity increased In contrast, the afferent pulse of the cardiac vagus is attenuated, and the slowing of the reflexive heart rate due to the pressurized stimulus is significantly reduced.