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本文对五个木材加工厂进行了调查,木尘浓度为4.5～9.6mg/m~3,游离SiO_2含量<5.0%。对301名木尘作业工人胸部X线检查,检出符合Ⅰ期尘肺X线改变者9例,其平均接尘工龄达29.1年。对107名木尘作业工人进行肺通气功能测定,肺功能改变轻微。对五种木尘用大鼠进行致肺纤维化实验,仅见粉尘细胞灶,未见胶原纤维增生。可以认为,木尘致纤维化作用较弱,胸片上小阴影的病理基础有待研究,接触木尘浓度<10mg/m~3发生尘肺的可能性较小。
In this paper, five wood processing plants were investigated, the wood dust concentration of 4.5 ~ 9.6mg / m ~ 3, free SiO2 content of <5.0%. Chest X-ray examination of 301 wood dust workers, detected in line with stage I pneumoconiosis X-ray changes in 9 cases, the average service life of dust up to 29.1 years. Pulmonary ventilation tests were performed on 107 workers exposed to wood dust, with minor changes in lung function. Pulmonary fibrosis experiments were performed on five kinds of wood dust in rats, only the dust cell foci, no collagen fiber proliferation. It can be assumed that the fibrinization effect of wood dust is weak, and the pathological basis of the small shadow on the chest radiograph is yet to be studied. There is less possibility of exposure to pneumoconiosis with dust concentration <10 mg / m3.