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采用家兔失血性休克模型，测定红细胞膜脂的流动性，膜脂区微粘度及相变温度。结果说明失血性休克后红细胞膜的荧光偏振度（Ｐ）升高，从０．２６０±０．０２０升至０．２８９±０．０１５（Ｐ＜０．０１），膜流动性降低。膜脂双层分子排列有序性增加。相变温度伤前为１７．５℃，低血压３ｈ后相变温度为２４．５℃，分子活化能升高。说明失血性休克后红细胞膜从相对比较流动变为相对固化，细胞膜更趋于凝胶相。
Rabbit hemorrhagic shock model was used to determine the fluidity of membrane lipids, the microviscosity of membrane lipid and the phase transition temperature. The results showed that the fluorescence polarization (P) of erythrocyte membrane increased from 0.260 ± 0.020 to 0.289 ± 0.015 (P <0.01) after hemorrhagic shock, and the membrane fluidity decreased. Membrane lipid bilayer molecules arranged orderly increase. Phase transition temperature pre-injury was 17.5 ℃, 3h after the phase transition temperature was 24.5 ℃, the molecular activation energy increased. It shows that after hemorrhagic shock, the erythrocyte membrane changes from relatively relatively fluid to relatively solidified, and the cell membrane tends to gel phase.