论文部分内容阅读
目的分析儿童急性一氧化碳中毒的临床特点及迟发性脑病的发生因素。方法对25例儿童急性一氧化碳中毒患儿的临床资料进行回顾性分析,分析儿童急性一氧化碳中毒的临床特点及迟发性脑病高危因素。结果(1)25例患儿中位年龄9.48岁,出现迟发性脑病3例,年龄9~11岁。(2)未发生迟发性脑病与迟发性脑病患儿昏迷时间、首次高压氧治疗时间比较差异均无统计学意义(P>0.05),迟发性脑病患儿抽搐次数多于未发生迟发性脑病患儿(P<0.05)。(3)迟发性脑病患儿白细胞计数显著高于未发生迟发性脑病患儿(P<0.01)。结论儿童一氧化碳中毒后抽搐发生次数及WBC明显升高,头颅影像学及脑电图异常越明显,发生迟发性脑病危险性越大。
Objective To analyze the clinical characteristics of children with acute carbon monoxide poisoning and the occurrence of delayed encephalopathy. Methods The clinical data of 25 children with acute carbon monoxide poisoning were analyzed retrospectively to analyze the clinical features and risk factors of delayed encephalopathy in children with acute carbon monoxide poisoning. Results (1) The median age of 25 children was 9.48 years. There were 3 cases of delayed encephalopathy, aged 9 to 11 years old. (2) There was no significant difference in the duration of first episode of hyperbaric oxygen between children with delayed encephalopathy and no delayed encephalopathy (P> 0.05), the number of twitch in children with delayed encephalopathy was longer than that in children without delayed encephalopathy Children with encephalopathy (P <0.05). (3) The number of leukocyte in children with delayed encephalopathy was significantly higher than that in children without delayed encephalopathy (P <0.01). Conclusions The number of convulsions and WBC in children with carbon monoxide poisoning were significantly higher than those before. The abnormal craniocerebral imaging and EEG were more obvious, and the risk of delayed encephalopathy was greater.