论文部分内容阅读
本文报道105例聋哑儿童的脑干听觉诱发电位测定(BAEP)。其中以氨基糖甙类抗生素药物中毒致聋为主89侧(80%以上),BAEP 表现为周围听神经损伤(Ⅰ,Ⅱ波消失)。而脑炎后遗症 BAEP 的Ⅰ—Ⅴ波各波潜伏期与正常对照组无明显差异。这提示氨基糖甙类抗生素致聋主要损伤耳蜗;而脑炎后遗症则损伤中枢。周围性损伤的治愈机会较大。
This article reports the brainstem auditory evoked potentials (BAEP) in 105 deaf children. Among them, aminoglycoside drug poisoning induced deafness 89 side (80%), BAEP showed peripheral auditory nerve injury (Ⅰ, Ⅱ wave disappeared). However, the latency of Ⅰ-Ⅴ waves in BAEP of encephalitis sequelae had no significant difference with that of the normal control group. This suggests that aminoglycoside-induced deafness primarily affects the cochlea, whereas encephalitis sepsis damages the center. Peripheral damage is more likely to be cured.