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镉在肾皮质中的过量蓄积可引起肾脏损害。实验证明,肾皮质中的镉达200—400微克/克(湿重)时,会出现肾功能和肾形态学的改变。估计镉作业工人肾皮质中镉的临界水平为200微克/克(湿重),因此预防肾皮质中镉接近临界水平极为重要。为此,作者进行动物实验来阐明长期接触镉时血镉和尿镉的意义,以寻求简便易行的评价肾损危险性的生物学指标。
Excessive accumulation of cadmium in the renal cortex can cause kidney damage. Experiments show that renal cortex cadmium up to 200-400 micrograms / gram (wet weight), there will be changes in kidney function and kidney morphology. It is estimated that the critical level of cadmium in renal cortex of cadmium workers is 200 μg / g (wet weight), so it is important to prevent cadmium in the renal cortex from approaching critical levels. To this end, the authors conducted animal experiments to clarify the significance of long-term exposure to cadmium and cadmium in cadmium in order to seek a simple and easy to assess the biological indicators of renal damage risk.