在一项新的研究中,来自日本大阪大学免疫学前沿研究中心(IFReC)的研究人员报道了一类新的他们称之为SatM的单核细胞。针对小鼠的研究表明SatM可能是导致组织纤维化的原因,而且成为一种治疗很少有有效疗法的疾病的新药物靶标。相关研究结果发表在Nature期刊上,论文标题为“Identification of an atypical monocyte and committed progenitor involved in fibrosis”。纤维化组织(fibrosis)是一种瘢痕组织,如果不加以控制的话,可能导致有害的组织增厚。尽管已知组织纤维化是由激活 In a new study, researchers from the Immunology Frontier Research Center (IFReC) at Osaka University in Japan reported a new class of monocytes called SatMs. Studies in mice suggest that SatM may be the cause of tissue fibrosis and become a new drug target for the treatment of diseases with few effective treatments. Relevant research results published in the Nature journal, the paper entitled “Identification of an atypical monocyte and committed progenitor involved in fibrosis ”. Fibrosis is a type of scar tissue that, if not controlled, can lead to thickening of unwanted tissue. Although it is known that tissue fibrosis is activated