作者对110例低血钠性中心性桥脑脱髓鞘(CPM)患者进行回顾性研究。低血钠分急性和慢性,低血钠引起的中枢神经系统症状常取决于其病因及急慢程度,由于烦喝排水调整受限、糖皮质激素缺乏,或因手术后应激、疼痛和合用多种药物(痛痉宁、噻嗪类利尿剂、前列腺素合成酶抑制剂)所致的抗利尿激素分泌失调综合症(SIADH)都可引起低血钠。嗜酒病人由于低盐和食物摄入减少,使肾脏内渗透性活性粒子(钠、尿素)降低而导致水排清受限。因自由水排清减少依其水的负荷情况而发展成急性或慢性低血钠。急性低血钠(血钠低于125mEq/l)由于急性水中毒而引起精神错乱、癫痫发作、假性球麻痹、Cheyne-Stoke 氏吸吸、嗜睡或深昏 The authors retrospectively studied 110 patients with hyponatremic central pontine demyelination (CPM). Hypoglycemic points acute and chronic, hyponatremia, central nervous system symptoms often depend on their etiology and degree of urgency, due to irritable drainage regulation is limited, glucocorticoid deficiency, or due to postoperative stress, pain and combined Many drugs (pain spasm, thiazide diuretics, prostaglandin synthase inhibitors) induced by anti-diuretic hormone syndrome (SIADH) can cause hyponatremia. Patients with alcohol abuse due to low salt and reduced food intake, so that the permeability of the kidneys within the active particles (sodium, urea) resulting in reduced water clearance. Acute or chronic hyponatremia develops due to the reduction of free water due to its water load. Acute hyponatremia (hyponatremia less than 125 mEq / l) Insane, seizures, pseudobulbar palsy, Cheyne-Stoke’s sucking, lethargy or drowsiness due to acute water poisoning