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血小板活性增高、前列环素(PGI_2)—凝血噁烷A_2(TXA_2,血栓素A_2)代谢失常及纤溶活性异常在冠心病发生和发展中,均有重要作用。钙拮抗剂抑制血小板聚集和影响TXA_2/PGI_2平衡,可能是其对冠心病治疗作用机制之一。
Increased platelet activity, abnormal metabolism of PGI_2-thromboxane A_2 (TXA_2, thromboxane A_2) and abnormal fibrinolytic activity play an important role in the occurrence and development of coronary heart disease. Inhibition of platelet aggregation of calcium antagonists and TXA_2 / PGI_2 balance may be one of its mechanisms of treatment of coronary heart disease.