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观察60例无室性心律失常的冠心病人在潘生丁试验前、中、后系列信号平均心电图(SA-ECG)的变化,以探讨一过性心肌缺血与心室晚电位(VLP)的关系.其中,28例(1组)发生了一过性心肌缺血,32例(2组)未诱发心肌缺血.潘生丁试验前SA-ECG异常者14例(23%):QRS时限>115ms者4例,滤波后QRS终末部40ms均方根电压(RMS 40)异常者6例,二者均异常4例,两组间无显著差异(24%与17%,P>0.05).两组病例在潘生丁试验前、中、后QRS时限和RMS 40的变化无显著差异(P>0.05).且一过性缺血性ST段抬高与压低的病人间亦无显著差异,试验前SA-ECG异常的病人试验中、后仍异常,试验前SA-ECG正常的病人试验中、后仍正常.提示,冠心病人潘生丁诱发的一过性心肌缺血是一种特殊类型的缺血,其电生理变化和VLP无明显关系.
To observe the change of SA-ECG in 60 patients with non-ventricular arrhythmia before and after the dipyridamole test to investigate the relationship between transient myocardial ischemia and ventricular late potential (VLP). Myocardial ischemia was induced in 28 (group 1) and myocardial ischemia was not observed in 32 (group 2) 14 (23%) patients with abnormal SA-ECG before dipyridamole: QRS duration> 115 ms 4 For example, there were 6 cases with RMS 40 abnormalities in QRS terminal after filtering, 4 cases were abnormal in both groups, and there was no significant difference between the two groups (24% vs 17%, P> 0.05) There were no significant differences in QRS duration and RMS 40 before, during and after dipyridamole (P> 0.05), and there was no significant difference between patients with transient ischemic ST-segment elevation and depression before SA-ECG Abnormal patient test, the latter still abnormal, normal SA-ECG before the test in patients with normal test, suggesting that coronary heart disease patients with dipyridamole-induced transient myocardial ischemia is a special type of ischemia, the electricity Physiological changes and VLP no significant relationship.