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患者,女,25岁,1年前(2004年4月)无明显原因出现活动后四肢易疲劳,1个月以后出现左上肢无力,活动后明显,但不影响日常生活。2004年6月起左手笨拙,双下肢沉重感及发僵,以上下楼时明显,走平路尚好,症状进行性加重。2004年11月发觉小便有时排不尽。2004年 12月在北京宣武医院检查显示患者以双侧锥体束损害为主,无肌萎缩,按“脱髓鞘病”试用大剂量丙种球蛋白(0．4· kg-1·d-1×5 d)治疗后,自觉小便症状和下肢发僵稍好转, 其余症状无明显改善。次年1月在当地医院再次用大剂量丙种球蛋白(剂量疗程同前)治疗无效。3个月前(2005
Patient, female, 25 years old, one year ago (April 2004) No obvious causes Extremities were tired after the activity. After one month, the left upper extremity was weak. After the activity, it did not affect daily life. Left hand clumsy since June 2004, both lower extremities a sense of heavy and stiff, clear down the stairs above, go flat is still good, the symptoms worsened. November 2004 found that sometimes empty row of urine. Xuanwu Hospital in Beijing in December 2004 showed that patients with bilateral pyramidal tract lesions, no muscle atrophy, according to “demyelinating disease” trial of high-dose gamma globulin (0.4 · kg-1 · d-1 × 5 d) After treatment, symptoms of spontaneous urination and lower extremity stiffening slightly improved, the remaining symptoms no significant improvement. January next year in the local hospital once again with high-dose gamma globulin (dose course with the former) treatment is invalid. 3 months ago (2005