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幽门螺杆菌 (HP)感染后胃黏膜持续的炎症反应导致胃黏膜损伤 ,IL - 8(interleukin- 8)等细胞因子起重要作用 ,HP菌株 cag致病岛 (cag pathogenicity island,cag PAI)的一些基因、ice A基因及编码外膜蛋白的某些基因片段与 HP感染后 IL - 8的表达分泌及胃黏膜的损伤密切相关 ,深入研究两者关系有助于进一步阐明 HP的致病机制。
Continued inflammation of the gastric mucosa following Helicobacter pylori (HP) infection causes gastric mucosal injury, cytokines such as interleukin-8 (IL-8) play an important role, and some of the cag pathogenicity island (cag PAI) The gene, ice A gene and some gene fragments encoding the outer membrane protein are closely related to the secretion of IL - 8 and the damage of gastric mucosa after HP infection. It is helpful to further understand the pathogenesis of HP by further studying the relationship between them.