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妊娠肾病(nephropathia gravidarum)在原始著作中仅指妊娠诱发高血压(即先兆子痫和子痫)的肾病变,其病理特点为肾小球毛细血管内皮增生、肿胀及空泡形成,系膜细胞增生,致使部分毛细血管腔闭塞。近年来,将妊娠期间发生的肾脏病统称为妊娠肾病。由于篇幅所限,只能叙述以下几个问题。一、妊娠期的肾脏生理改变在妊娠早期即可见到肾长度增加(X线下约增加1cm),肾小球增大;同时伴有肾盏、肾盂及输尿管扩张,以右侧为著。这些改变直至分娩后12～16周才完全复原。妊娠期尿集合系统生理性扩张给临床带来一些麻烦,例如集合系统尿容量增加,使计时集尿的检验结果产生误差;对肾血浆流量测定、肾小球滤过率测定及雌三醇,17-酮(或羟)类固醇,肌酐及电
Nephropathia gravidarum in the original book refers only to nephropathy in pregnancy-induced hypertension (ie preeclampsia and eclampsia), pathological features of which are glomerular capillary endothelial hyperplasia, swelling and vacuolization, mesangial cell proliferation , Resulting in some of the capillary cavity occlusion. In recent years, the occurrence of kidney disease during pregnancy collectively referred to as pregnancy kidney disease. Due to space limitations, can only be described the following questions. First, the physiological changes of the kidney during pregnancy can be seen in early pregnancy to increase the length of the kidneys (X line increased by about 1cm), glomerular enlargement; accompanied by calyx, renal pelvis and ureteral dilatation to the right side. These changes did not fully recover until 12-16 weeks after childbirth. Physiological dilatation of the urine collection system in pregnancy brings some troubles to the clinic, for example, the urine volume of the collection system is increased to make the test result of the timed urine collection error; the determination of renal plasma flow, the determination of glomerular filtration rate and estriol, 17-keto (or hydroxy) steroids, creatinine and electricity