论文部分内容阅读
模拟煤烟型氟中毒地区农民燃煤方式,设高氟高硫组(氟化物与SO_2浓度分别为0.631和40.97 mg/m~3)、高氟低硫组(氟化物与SO_2浓度分别为0.583和5.086 mg/m~3),以及无燃煤污染的对照组(氟化物与SO_2浓度分别为0.008和0.191 mg/m~3), SD大鼠自然吸入中毒5个月。实验结果表明,中毒组大鼠均发生了氟斑牙,对照组牙齿无变化;实验组大鼠血清氟、尿氟、骨氟与牙齿氟均非常显著高于对照组。高氟高硫组大鼠的氟斑牙发生率高于高氟低硫组,牙齿病变表现严重,显示SO_2对大鼠的氟斑牙病变有加强作用。
In the high-fluorine and high-sulfur group (0.631 and 40.97 mg / m ~ 3 for fluoride and SO_2, respectively), the high-fluorine and low-sulfur group (the concentrations of fluoride and SO_2 were 0.583 And 5.086 mg / m ~ 3 respectively), and the control group without coal pollution (fluoride and SO_2 concentrations were 0.008 and 0.191 mg / m ~ 3, respectively). SD rats were naturally inhaled for 5 months. The experimental results showed that all the rats in the poisoning group developed dental fluorosis, while the teeth in the control group showed no change. The levels of serum fluoride, urinary fluoride, bone fluoride and dental fluorosis in the experimental group were significantly higher than those in the control group. The incidence of dental fluorosis in high-fluorine and high-sulfur group was higher than that in high-fluorine and low-sulfur group, and the dental lesion was serious. It showed that SO 2 had a strong effect on the dental fluorosis in rats.