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众所周知,慢性肾功能不全存在着蛋白质的代谢异常,有关其发病机制和临床意义目前尚未明了。对此,笔者总结我院1990年1月至1994年12月130例慢性肾功能不全住院病人的资料,按不同的临床类型和肾功能分期,分析其血清蛋白变化规律,试图为其发病机制,临床诊断,鉴别诊断及治疗提供一些依据。1 资料与方法1.1 诊断标准:按1992年原发性肾小球疾病专题座谈会提出的原发性肾小球疾病临床分型标准及慢性肾功能衰竭分期标准,本组130例中慢性肾炎68例、隐匿性肾炎7例、肾病综合征55例。肾功能正常期22例、代偿期31例、失代偿期25例、肾功能衰竭期19例和尿毒症期33例。
As we all know, chronic renal insufficiency exists protein metabolic abnormalities, its pathogenesis and clinical significance is not yet clear. In this regard, the author summarizes our hospital from January 1990 to December 1994 in 130 cases of chronic renal insufficiency inpatients, according to different clinical types and stages of renal function, analysis of the changes of serum proteins, trying to its pathogenesis, Clinical diagnosis, differential diagnosis and treatment to provide some basis. 1 Materials and Methods 1.1 Diagnostic criteria: According to the 1992 primary glomerular disease symposium proposed primary glomerular disease clinical classification criteria and chronic renal failure staging criteria, the group of 130 cases of chronic nephritis 68 For example, occult nephritis in 7 cases, nephrotic syndrome in 55 cases. 22 cases of normal renal function, decompensation in 31 cases, 25 cases of decompensation, renal failure in 19 cases and uremia in 33 cases.