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检测63例重症肌无力患者和50例正常健康人血浆乙酰胆碱酶(P-AchE)活性、红细胞乙酰胆碱酯酶(E-AchE)活性。结果,患者组的P-AchE平均活性和E-AchE平均活性皆显著高于对照组(P<0.001)。提示重症肌无力的发病机制除了自身免疫因素外,还有乙酰胆碱酯酶活性改变的因素。
The activity of acetylcholinesterase (P-AchE) and erythrocyte acetylcholinesterase (E-AchE) in 63 patients with myasthenia gravis and 50 normal controls were measured. As a result, the mean P-AchE activity and the average E-AchE activity of the patient group were significantly higher than those of the control group (P <0.001). Tip of the pathogenesis of myasthenia gravis In addition to autoimmune factors, there are changes in acetylcholinesterase activity.