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过去研究证明,在急性心肌梗塞时交感-肾上腺系统活性的增高,反映在尿排出的去甲肾上腺素、肾上腺素、多巴胺、香草扁桃酸(VMA)和甲氧基儿茶酚胺类增加。本文报告急性心肌梗塞患者尿中另一儿茶酚胺代谢物-3-甲氧-4-羟苯基乙二醇(MHPG)的排出量。作者研究的30例急性心肌梗塞患者中,男25例,女5例,平均年龄52.6岁(35-70岁)。其中13例无并发症,17例有并发症(心律不齐,传导异常和心力衰竭等)。患者最迟于心肌梗塞急性发作后24小时内开始收集24小时尿液,连续5天。每份尿液标本分别用荧光光谱法测定MHPG、去甲肾上
Past studies have shown that increased sympathetic-adrenal system activity in acute myocardial infarction is reflected in the increased urinary excretion of norepinephrine, epinephrine, dopamine, vanillylmandelic acid (VMA), and methoxycatecholamines. This article reports the excretion of another catecholamine metabolite, 3-methoxy-4-hydroxyphenylglycol (MHPG), in the urine of patients with acute myocardial infarction. Of the 30 patients with acute myocardial infarction studied, 25 were males and 5 were females, with an average age of 52.6 years (35-70 years). Of these, 13 had no complications and 17 had complications (arrhythmia, conduction abnormalities, heart failure, etc.). The patient started to collect urine for 24 hours within 24 hours after the acute attack of myocardial infarction for 5 consecutive days. Each urine sample was measured by fluorescence spectrometry MHPG, norepinephrine