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研究证实易损斑块出现的同时伴随着内皮功能紊乱、内皮细胞脱失以及炎症反应的存在。内皮受损后可引起炎症反应激活、单核细胞浸润和血管平滑肌细胞增生,进而促发动脉粥样硬化和再狭窄形成,故修复受损血管内皮、促使血管重新内皮化已经成为防止动脉粥样硬化及再狭窄领域的重要课题。与此同时,内皮祖细胞参与受损血管的重新内皮化,吸引了越来越多的关注。现就内皮祖细胞与易损斑块的研究进展进行综述。
Studies have shown that the presence of vulnerable plaque accompanied by endothelial dysfunction, endothelial cell loss and inflammation. Endothelial damage can cause inflammatory response activation, monocyte infiltration and vascular smooth muscle cell proliferation, thereby promoting the formation of atherosclerosis and restenosis, it is to repair the damaged vascular endothelium, vascular endothelial re-endothelialization has become to prevent atherosclerosis Sclerotherapy and restenosis in the field of important topics. At the same time, endothelial progenitor cells participate in the re-endothelialization of damaged blood vessels, attracting more and more attention. The research progress on endothelial progenitor cells and vulnerable plaque is reviewed.