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患者男性,34岁,某颜料厂烘磨车间操作工,接变。触硝酸铅、铬酸铅一年。平时经常感头痛、头昏、乏力,近因持续性腹痛阵发性加剧一天,伴恶心、呕吐急诊入院。入院时腹部及神经系统未见阳性体征。验尿铅2.55mg/L,δ—ALA74mg/L、尿棕色素(++++)点彩红细胞160只/50视野、碱粒凝集试验18％。诊断为慢性中度铅中毒。 入院后即行驱铅治疗。驱铅药物为依地酸二钠钙(EDTA)0.25g,静脉注射,每日二次。每周用药三天,停药四天为一疗程。共用药二个疗程。于第二疗程始查脑电图示中度不正常。脑电图特征主要
Male patient, 34 years old, a paint factory bakery shop operator, then change. Touch lead nitrate, lead chromate one year. Often the usual headache, dizziness, fatigue, exacerbation of persistent abdominal pain due to paroxysmal one day, with nausea and vomiting emergency admission. There were no positive signs in the abdomen and nervous system at admission. Urine lead 2.55mg / L, δ-ALA74mg / L, urine brown pigment (++++) 160 red blood cells / 50 field of view, alkali agglutination test 18%. Diagnosed as chronic moderate lead poisoning. Lead to lead treatment after admission. Driven lead drugs for edetate disodium calcium (EDTA) 0.25g, intravenous injection, twice daily. Medication for three days a week, withdrawal for four days for a course of treatment. Medication two courses. EEG in the second course of treatment showed moderate abnormalities. EEG features the main