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本文研究了富含甘油三酯(TG)的脂蛋白促使主动脉中膜平滑肌细胞(SMC)脂类堆积,从而转变为泡沫细胞的可能机制。发现正常人极低密度脂蛋白(VLDL)与兔主动脉SMC温育,细胞内脂类增加。巨噬细胞(Mq_φ)明显促进这一作用。
This study investigated the possible mechanisms by which triglyceride (TG) -rich lipoproteins promote lipid accumulation in the aortic tunica intima and to foam cells. Found that normal human very low density lipoprotein (VLDL) and rabbit aortic SMC incubation, increased intracellular lipid. Macrophages (Mq_φ) significantly promote this effect.