前言哮喘从病理上来说其特征是炎症细胞如嗜酸性细胞和淋巴细胞浸润至气道壁、气道上皮细胞损害;粘膜水肿和气道微血管渗漏。由于平滑肌收缩和气道水肿而引起的气道阻塞。此外,哮喘者也表现为支气管高敏状态。有增加的事实证明,哮喘的气道阻塞和支气管高反应性是由于哮喘时炎症过程的结果。鉴于哮喘机理概念的新近进展,我们将目前应用于治疗哮喘的药物分为支气管扩张药(如β_2-肾上腺素激动剂)和抗炎药(如糖皮质激素)。 Preface Asthma is pathologically characterized by infiltration of inflammatory cells such as eosinophils and lymphocytes into the airway wall, airway epithelial cell damage, mucosal edema, and airway microvascular leakage. Airway obstruction due to smooth muscle contraction and airway edema. In addition, asthma also showed bronchial hypersensitivity. There is an increase in the fact that airway obstruction and bronchial hyperreactivity in asthma are the result of inflammatory processes at asthma. Given the recent advances in the concept of asthma mechanisms, we classify the drugs currently used to treat asthma as bronchodilators (such as β_2-adrenergic agonists) and anti-inflammatory drugs (such as glucocorticoids).