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正党人在白天期间的房水排出率为2.75±0.63μl/min。正常范围(95%)为1.8至4.3μl/min。房水排出有生理性周期,早上高,下午稍低,睡眠时最低,约为早上的一半。10岁后,每10年房水排出率下降3.2%。男女无明显区别。有3类治疗青光眼的药物可抑制房水生成:碳酸酐酶抑制剂,β-肾上腺素能对抗剂,α_2-选择性肾上腺素能促效剂。噻吗心安在白天有很强的抑制房水生成的作用,而在睡眠时作用消失。乙酰唑胺和apraclonidine 可降低睡眠者的房水生成率。β-肾上腺素能对抗剂和α_2-肾上腺素能促效剂在急性治疗中没有相加作用。β-肾上腺素能对抗剂和碳酸酐酶抑制剂有部分相加作用。对于噻吗心安为长期使用其作用下降,当停药后其作用可逐渐恢复。房水排出率对眼压调节性变化并不敏感。
Members of the party during the day of atrial water discharge rate of 2.75 ± 0.63μl / min. The normal range (95%) is 1.8 to 4.3 μl / min. Room water discharge physiological cycles, high morning, slightly lower afternoon, the lowest sleep, about half of the morning. After 10 years of age, aqueous humor discharge dropped by 3.2% every 10 years. No significant difference between men and women. There are three classes of drugs that treat glaucoma that inhibit aqueous humor formation: carbonic anhydrase inhibitors, β-adrenergic antagonists, and α 2-selective adrenergic agonists. Timolol in the daytime there is a strong inhibitory effect of aqueous humor, and the role disappeared in sleep. Acetazolamide and apraclonidine reduce the rate of aqueous humor in the sleepers. Beta-adrenergic antagonists and alpha-2-adrenergic agonists have no additive effect in acute treatment. Beta-adrenergic antagonists and carbonic anhydrase inhibitors partially additive. For timolol for long-term use of its role in the decline, when the withdrawal of its role can be gradually restored. Aqueous discharge rate is not sensitive to changes in intraocular pressure.