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Two mutants with rolled leaves, temporally designated as rl3(t)-1 and rl3(t)-2, were served for exploring the mechanism underlying the rolled leaf characteristic. Except for having typical rolled leaves, the plant heights and panicle lengths of rl3(t)-1 and rl3(t)-2 significantly decreased, and the seed-setting rate also decreased when compared with wild type 93-11. Cytological analysis suggested that the rolled leaf phenotype might be caused by the changes of number and size of bulliform cells. Genetic analysis indicated rl3(t)-1 is allelic to rl3(t)-2, and controlled by a recessive gene. Gene mapping result indicated that RL3(t) gene resided in a 46-kb long region governed by the sequence tag site markers S3-39 and S3-36 on rice chromosome 3. The result provides an important clue for further cloning the RL3(t) and understanding the mechanism of rice leaf development.
Two mutants with rolled leaves, temporally designated as rl3 (t) -1 and rl3 (t) -2, were served for exploring the mechanism underlying the rolled leaf characteristic. Except for having typically rolled leaves, the plant heights and panicle lengths of rl3 (t) -1 and rl3 (t) -2 significantly decreased, and the seed-setting rate also decreased when compared with wild type 93-11. Cytological analysis suggested that the rolled leaf phenotype might be caused by the changes of number and size of bulliform cells. Genetic analysis indicated rl3 (t) -1 is allelic to rl3 (t) -2, and controlled by a recessive gene. Gene mapping result indicated that RL3 (t) gene resided in a 46-kb long region governed by the sequence provides site markers S3-39 and S3-36 on rice chromosome 3. The result provides an important clue for further cloning the RL3 (t) and understanding the mechanism of rice leaf development.