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男性,60岁,1991年6月24日入院。患者于入院前一天在田间劳动时突然出现两眼视物不清,约半小时后双眼视力恢复正常。12小时后再次出现双眼视物不清,伴右颞部轻度跳痛,无恶心、呕吐及眩晕,左侧肢体活动不灵,无意识障碍、癲痫发作及尿便障碍。高血压病史5年。入院时查体:BF22/14kPa,神清语明,球结膜无充血、水肿,眼位无异常,视力眼前手动,瞳孔等大同圆,对光反应灵敏,左侧鼻唇沟略浅,伸舌尚居中,左手握力弱,左下肢肌力4级,左侧偏身痛觉减退,左侧腹壁反射及提睾反射消失,双侧掌颏反射(+),左侧Babindki氏征(+)。无项强,腰穿脑脊液无色澄清透明,压力2.0kPa,Pandy氏反应(+),WBC9×10~6/L,未见到皱缩红细胞,糖4.92mmol/L。蛋白0.62g/
Male, 60 years old, admitted to hospital on June 24, 1991. Patients in the field one day before admission suddenly appeared in both eyes blurred vision, about half an hour later, binocular vision returned to normal. Twelve hours later, his eyes were palpable again with mild visual acuity, slight right throbbing in the right temporal area, nausea, vomiting and dizziness, impaired left limb movement, unconsciousness, seizures and urinary problems. Hypertensive history of 5 years. Admission examination: BF22 / 14kPa, Shen Qing language, conjunctival hyperemia, edema, no abnormalities in the eye position, eyesight before hand, pupil and other Datong round, sensitive to light, the left slightly shallow nasolabial fold, tongue extension Still living in the left hand grip weak, left lower extremity muscle strength 4, left partial body pain decreased, left abdominal wall and cremaster reflex disappeared, bilateral palmar reflex (+), left Babindki’s sign (+). No item is strong, lumbar cerebrospinal fluid was colorless and transparent, pressure 2.0kPa, Pandy’s reaction (+), WBC9 × 10 ~ 6 / L, no shrinkage erythrocytes and sugar 4.92mmol / L. Protein 0.62 g /