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嗜酸细胞(EOS)是哮喘慢性炎症中的关键效应细胞。IL5、IL3能促进EOS在骨髓的分化、成熟,使机体的EOS产生增多,并促使EOS在支气管肺组织的聚集及活化。哮喘患者外周血、支气管粘膜、支气管肺泡灌洗液(BALF)中IL5明显升高[1]。IL3
Eosinophils (EOS) are key effector cells in chronic inflammation of asthma. IL 5, IL 3 can promote the differentiation and maturation of EOS in the bone marrow, so that the body produces increased EOS, and promote EOS in bronchial lung tissue aggregation and activation. Asthma patients with peripheral blood, bronchial mucosa, bronchoalveolar lavage fluid (BALF) IL 5 was significantly increased [1]. IL 3