论文部分内容阅读
1983年Mazur[1]首次提出胃肠道间质瘤(gastrointestinalstromal tumors,GISTs)的诊断以后,作为一种独立的消化道疾病一直是研究热点。GIST可发生自食管至肛门的胃肠道全长范围,瘤细胞显示CD117及CD34有较高的阳性表达率[2,3]。血管内皮生长因子(VEGF)及其受体(VEGFR-2)被认为是
After Mazur [1] first proposed the diagnosis of gastrointestinal gastrointestinal stromal tumors (GISTs) in 1983, it has been a research hot spot as an independent gastrointestinal disease. GIST can occur over the full length of the gastrointestinal tract from the esophagus to the anus, and tumor cells show a higher positive rate of CD117 and CD34 [2,3]. Vascular endothelial growth factor (VEGF) and its receptor (VEGFR-2) are considered