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类风湿性关节炎是一种常见的慢性多发性关节炎,属于结缔组织疾病。1 病理分析 初为滑膜充血、水肿和炎细胞浸润。继而滑膜上有富于血管的肉芽组织生长,呈翳状被覆于关节软骨表现。炎细胞中的白细胞产生多量胶原酶,引起关节软骨、关节囊和肌腱等组织破坏;中性白细胞在吞噬类风湿因子的同时,本身也被破坏。胞浆中的溶酶释放出大量水解酶,导致组织进一步破坏,炎性组织逐渐纤维化或骨化。2 临床资料 对69名类风湿关节炎病人跟骨侧位片观察,男21人,女48人;年龄20～40岁。类风湿性因子检查,阳性者58人,弱阳性者11人。
Rheumatoid arthritis is a common chronic polyarthritis, which is a connective tissue disease. A pathological analysis of synovial hyperemia, edema and inflammatory cell infiltration. Then the synovial vascular growth of granulation tissue, was 翳 shaped coating in the performance of articular cartilage. Leukocytes in inflammatory cells produce large amounts of collagenase, causing destruction of articular cartilage, joint capsule and tendon tissue; neutrophils phagocytosis of rheumatoid factor itself, but also destroyed. Cytoplasm lysosidase release a large number of hydrolases, leading to further destruction of tissue, inflammatory tissue gradually fibrosis or ossification. 2 Clinical data of 69 patients with rheumatoid arthritis lateral calcaneal observation, 21 males and 48 females; aged 20 to 40 years. Rheumatoid factor test, 58 were positive, 11 were weakly positive.