过氧化损伤是糖尿病慢性并发症发病的一个十分重要环节，而过氧化酶尤其是超氧歧化酶（ＳＯＤ）的代谢异常为这一损伤发生和发展的一个重要机制［１］。为了探讨过氧化损伤与糖尿病微血管并发症一糖尿病肾病肾功能损伤之间的关系，本文对３５例Ⅱ型无糖尿病肾病激６０例Ⅱ型糖 Peroxidative damage is a very important part of the pathogenesis of chronic complications of diabetes, and the abnormal metabolism of peroxidase, especially superoxide dismutase (SOD) is an important mechanism of the occurrence and development of this injury [1]. In order to investigate the relationship between oxidative damage and diabetic microangiopathy and diabetic nephropathy renal injury, this article 35 patients with type II non-diabetic nephropathy stimulated type 60 sugar