论文部分内容阅读
目的 :探讨一氧化氮在功能性消化不良发病机制中的作用。方法 :应用硝酸还原酶法测定 2 0例正常人 ,6 0例功能性消化不良患者血清一氧化氮水平。结果 :功能性消化不良患者血清一氧化氮水平为 (75 .5 8±18.4 2 ) μmo1/L ,明显高于正常对照组 ,(46 .0 7± 19.0 9) μmol/L(P <0 .0 1)。结论 :内源性一氧化氮可能参与功能性消化不良的发病机制。
Objective: To investigate the role of nitric oxide in the pathogenesis of functional dyspepsia. Methods: Nitric acid reductase method was used to determine serum nitric oxide level in 20 normal controls and 60 patients with functional dyspepsia. Results: The level of serum nitric oxide in patients with functional dyspepsia was (75.58 ± 18.4 2) μmo1 / L, which was significantly higher than that in the control group (46.0 ± 19.0 9) μmol / L (P <0. 0 1). Conclusion: Endogenous nitric oxide may be involved in the pathogenesis of functional dyspepsia.