【摘 要】
：
胃癌发生的分子基础与其他肿瘤一样是原癌基因的活化或抑癌基因的失活,导致某些细胞的分化不良和增殖失控而致。目前,对癌基因和抑癌基因的研究已成为肿瘤研究的热点,而P16基
【机 构】
：
皖南医学院附属弋矶山医院 安徽芜湖241000
论文部分内容阅读
胃癌发生的分子基础与其他肿瘤一样是原癌基因的活化或抑癌基因的失活,导致某些细胞的分化不良和增殖失控而致。目前,对癌基因和抑癌基因的研究已成为肿瘤研究的热点,而P16基因作为新发现的抑癌基因又倍受人们的关注。1 P16基因的结构与功能 1993年Serrano等[1] 发现并分离
The molecular basis of gastric cancer, like other tumors, is the activation of protooncogenes or the inactivation of tumor suppressor genes, resulting in poor differentiation of some cells and uncontrolled proliferation. At present, the research on oncogenes and tumor suppressor genes has become a hot topic in cancer research, and P16 gene as a newly discovered tumor suppressor gene has drawn much attention. 1 P16 gene structure and function In 1993 Serrano et al [1] found and isolated
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