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心力衰竭(简称心衰)是指因心肌收缩性减弱,以致在静息或一般体力活动时不能输出足够量的血液,满足机体组织代谢的需要所引起的病理过程。心衰是心脏病的常见并发症,且为心脏病的主要致死原因。近年来随着基础与临床研究不断深入,特别是对控制心肌作功和心肌力学的知识有了很大进展,对心衰的病理生理改变有了某些新的认识。本文侧重心衰时机械功能、神经体液机制和心肌膜功能等方面的变化,复习文献介绍于下。
Heart failure (referred to as heart failure) is due to myocardial contractility weakened, resulting in rest or general physical activity can not output a sufficient amount of blood to meet the needs of the body tissue metabolism caused by the pathological process. Heart failure is a common complication of heart disease and is the leading cause of death from heart disease. In recent years, with the deepening of basic and clinical research, especially for the control of myocardial function and myocardial mechanics knowledge has made great progress, the pathophysiology of heart failure has some new understanding. This article focuses on changes in mechanical function, neurohumoral mechanism and myocardial membrane function in heart failure, review the literature introduced in the next.